ISPP 2015

Career Opportunities in Pharmacy
3D Medical Animation Congestive Heart Failure Animation



despite ongoing advances in the management of chronic heart failure patient outcomes for acutely decompensated disease have not improved across Canada patients hospitalized for heart failure continue to suffer frequent complications and 16.5% die during their initial hospitalization an examination of the pathophysiology of acute decompensated heart failure identifies promising new avenues for treatment during heart failure the heart cannot circulate enough blood abnormal cardiac filling pressures to meet the metabolic needs of the brain kidneys heart and other vital organs the body responds by releasing neurohormones certain neurohormones help maintain blood pressure but over time they cause heart failure to worsen one of these neuro hormones is weaning released by the kidneys in response to decrease the fusion and increased sympathetic activity greenin activates the renin-angiotensin-aldosterone system or RAAS by cleaving angiotensinogen in the liver to produce angiotensin 1 which is further converted to angiotensin 2 in the lungs angiotensin 2 binds to blood vessel walls causing vasoconstriction angiotensin ii also stimulates release event of feeling from endothelial cells angiotensin ii and endothelium act on vascular smooth muscle causing constriction of the coronary arteries and systemic arteries and veins while vasoconstriction helps maintain blood pressure it can also have maladaptive effects for example the weakened heart must pump harder against increased resistance in the systemic arteries while constriction of the coronary arteries further compromises the myocardium supply of oxygen venous constriction also contributes to a displacement of fluid into the lungs and other tissues angiotensin ii stimulates the adrenal glands to release and östra aldosterone causes the kidneys to reabsorb sodium into a blood stream with it water sodium and water attention cause a state of fluid overload that is exacerbated by venous constriction gaseous exchange in the lungs is inhibited by Palmeri edema buildup of fluid in and around the lungs increases the physical work of breathing patients with acute left-sided heart failure feel short of breath and fatigue when right-sided heart failure occurs fluid accumulates in the liver and legs apart from the activity of the RAAS the body tries to maintain blood pressure using other mechanisms such as releasing epinephrine from the adrenal glands and norepinephrine from sympathetic nerve endings these catecholamines increase heart rate and contractility in addition to causing arterial and venous constriction over time chronic volume overload and the activity of angiotensin 2 aldosterone and endothelium stimulate pathologic cardiac remodelling the interstitial fibrosis and myocyte apoptosis associated with cardiac remodelling decrease heart wall elasticity further inhibiting the heart's ability to relax and pump effectively in summary the neuro hormonal response to heart failure causes vasoconstriction fluid retention and increased heart rate and contractility in turn these processes initiate a vicious cycle of further myocardial injury and escalating heart failure fortunately additional neuro hormonal responses modulate the activity of the IaaS endothelium epinephrine and norepinephrine

33 thoughts on “3D Medical Animation Congestive Heart Failure Animation

  1. Would be a better video if there wasn't all this loud crashing and banging. I thought it was coming from somewhere else so I paused the video.

    The disrupting sound has nothing to the video; better to leave it out.

  2. Just do a 48 hour dry fast every week and eat one meal a day. This will eliminate and or reversed all of this by indefinitely clearing your arteries and cleaning the body overall

  3. So I was diagnosed with CHF at the age of 32. It was found that I was born with an enlarged heart. The size of a 6 month old baby at 1 day old. I'm researching like crazy to find out how to cure it without the need for surgery. The docs have me taking Bumetanide 2mg which makes me urinate like crazy but I have problems with the limited fluid intake I'm suppose to be on. I found that there is a machine that can safely remove salt and access fluid from the body. I saw that a woman lost 30 Ibs of fluid in 2 days. I try going to the gym and taking my meds but the weight just fluctuates constantly. Under hospital care I lost almost 30 in 5 days but on my own the weight comes and goes. There has to be a way to cure it. If anyone out there knows of another direction I should take, please let me know.

  4. Great Video! Forgive me for the intrusion, I would love your opinion. Have you heard the talk about – Trentvorty Irreplaceable Doctor Theorem (should be on google have a look)? It is an awesome one off guide for curing heart disease without the hard work. Ive heard some great things about it and my GF finally got great results with it.

  5. This kid clearly doesn't like having her calf muscles stretched.  Unfortunately, it's something she has to put up with.  Years from now she will be saying she was happy to have it.

  6. Fantastic presentation. Would love the video for classroom use with nursing students and in the telemetry units.  Thank you for sharing, your all brilliant . . . 

  7. We in need to antagonist these changes that occur as a consequence of heart failure , however. Hypercouaglable state and inflammatory markers must be understood to with properly , thank you do much indeed

  8. My mother died two years ago from Congestive Heart Failure. She just dropped in the kitchen and never came to. She was 43. I hope she never realized a thing and went peacefully. I just want to understand this better.

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